| THE MECHANISMS OF
DARWINISM
According to the theory of evolution, living things
came into existence by means of coincidences, and developed further
as a consequence of coincidental effects. Approximately 3.8 billion
years ago, when no living organisms existed on earth, the first
simple single-celled organisms (prokaryotes) emerged. Over time,
more complex cells (eukaryotes) and multicellular organisms came
into being. In other words, according to Darwinism, the forces of
nature built simple inanimate elements into highly complex and flawless
designs.
In evaluating this claim, one should first consider
whether such forces in fact exist in nature. More explicitly, are
there really natural mechanisms which can accomplish evolution according
to the Darwinian scenario?
The neo-Darwinist model, which we shall take as the
mainstream theory of evolution today, argues that life has evolved
through two natural mechanisms: natural selection and mutation.
The theory basically asserts that natural selection and mutation
are two complementary mechanisms. The origin of evolutionary modifications
lies in random mutations that take place in the genetic structures
of living things. The traits brought about by mutations are selected
by the mechanism of natural selection, and by this means living
things evolve. However, when we look further into this theory, we
find that there is no such evolutionary mechanism. Neither natural
selection nor mutations can cause different species to evolve into
one another, and the claim that they can is completely unfounded.
Natural Selection
The concept of natural selection was the basis of Darwinism.
This assertion is stressed even in the title of the book in which
Darwin proposed his theory: The Origin of Species, by means of Natural
Selection…
Natural selection is based on the assumption that in
nature there is a constant struggle for survival. It favors organisms
with traits that best enable them to cope with pressures exerted
by the environment. At the end of this struggle, the strongest ones,
the ones most suited to natural conditions, survive. For example,
in a herd of deer under threat from predators, those individuals
that can run fastest will naturally survive. As a consequence, the
herd of deer will eventually consist of only fast-running individuals.
However, no matter how long this process goes on, it
will not transform those deer into another species. The weak deer
are eliminated, the strong survive, but, since no alteration in
their genetic data takes place, no transformation of a species occurs.
Despite the continuous processes of selection, deer continue to
exist as deer.
The deer example is true for all species.
In any population, natural selection only eliminates those weak,
or unsuited individuals who are unable to adapt to the natural conditions
in their habitat. It does not produce new species, new genetic information,
or new organs. That is, it cannot cause anything to evolve. Darwin,
too, accepted this fact, stating that "Natural selection can do
nothing until favourable individual differences or variations occur."7
That is why neo-Darwinism had to add the mutation mechanism as a
factor altering genetic information to the concept of natural selection.
We will deal with mutations next. But before
proceeding, we need to further examine the concept of natural selection
in order to see the contradictions inherent in it.
A Struggle for Survival?

Darwin had been influenced by Thomas Malthus when he developed
his thesis of the struggle for life. But observations and
experiments proved Malthus wrong. |
The essential assumption of the theory of natural selection
holds that there is a fierce struggle for survival in nature, and
every living thing cares only for itself. At the time Darwin proposed
this theory, the ideas of Thomas Malthus, the British classical
economist, were an important influence on him. Malthus maintained
that human beings were inevitably in a constant struggle for survival,
basing his views on the fact that population, and hence the need
for food resources, increases geometrically, while food resources
themselves increase only arithmetically. The result is that population
size is inevitably checked by factors in the environment, such as
hunger and disease. Darwin adapted Malthus's vision of a fierce
struggle for survival among human beings to nature at large, and
claimed that "natural selection" is a consequence of this struggle.
Further research, however, revealed
that there was no struggle for life in nature as Darwin had postulated.
As a result of extensive research into animal groups in the 1960s
and 1970s, V. C. Wynne-Edwards, a British zoologist, concluded that
living things balance their population in an interesting way, which
prevents competition for food. Animal groups were simply managing
their population on the basis of their food resources. Population
was regulated not by elimination of the weak through factors like
epidemics or starvation, but by instinctive control mechanisms.
In other words, animals controlled their numbers not by fierce competition,
as Darwin suggested, but by limiting reproduction.8
Even plants exhibited examples of
population control, which invalidated Darwin's suggestion of selection
by means of competition. The botanist A. D. Bradshaw's observations
indicated that during reproduction, plants behaved according to
the "density" of the planting, and limited their reproduction if
the area was highly populated with plants.9 On
the other hand, examples of sacrifice observed in animals such as
ants and bees display a model completely opposed to the Darwinist
struggle for survival.
In recent years, research has revealed
findings regarding self-sacrifice even in bacteria. These living
things without brains or nervous systems, totally devoid of any
capacity for thought, kill themselves to save other bacteria when
they are invaded by viruses.10
These examples surely invalidate the basic assumption
of natural selection-the absolute struggle for survival. It is true
that there is competition in nature; however, there are clear models
of self-sacrifice and solidarity, as well.
Observation and Experiments
Apart from the theoretical weaknesses mentioned above,
the theory of evolution by natural selection comes up against a
fundamental impasse when faced with concrete scientific findings.
The scientific value of a theory must be assessed according to its
success or failure in experiment and observation. Evolution by natural
selection fails on both counts.
Since Darwin's time, there has not been a single shred
of evidence put forward to show that natural selection causes living
things to evolve. Colin Patterson, the senior paleontologist at
the British Museum of Natural History in London and a prominent
evolutionist, stresses that natural selection has never been observed
to have the ability to cause things to evolve:
No one has ever produced a
species by the mechanisms of natural selection. No one has ever
got near it, and most of the current argument in neo-Darwinism is
about this question.11
Pierre-Paul Grassé, a well-known French zoologist and
critic of Darwinism, has these words to say in "Evolution and Natural
Selection," a chapter of his book The Evolution of Living Organisms.
The "evolution in action" of J. Huxley
and other biologists is simply the observation of demographic facts,
local fluctuations of genotypes, geographical distributions. Often
the species concerned have remained practically unchanged for hundreds
of centuries! Fluctuation as a result of circumstances, with prior
modification of the genome, does not imply evolution, and we have
tangible proof of this in many panchronic species [i.e. living fossils
that remain unchanged for millions of years].12
A close look at a few "observed examples of natural
selection" presented by biologists who advocate the theory of evolution,
would reveal that, in reality, they do not provide any evidence
for evolution.
The True Story of Industrial Melanism
When evolutionist sources are examined, one inevitably
sees that the example of moths in England during the Industrial
Revolution is cited as an example of evolution by natural selection.
This is put forward as the most concrete example of evolution observed,
in textbooks, magazines, and even academic sources. In actuality,
though, that example has nothing to do with evolution at all.
Let us first recall what is actually said: According
to this account, around the onset of the Industrial Revolution in
England, the color of tree barks around Manchester was quite light.
Because of this, dark-colored moths resting on those trees could
easily be noticed by the birds that fed on them, and therefore they
had very little chance of survival. Fifty years later, in woodlands
where industrial pollution has killed the lichens, the bark of the
trees had darkened, and now the light-colored moths became the most
hunted, since they were the most easily noticed. As a result, the
proportion of light-colored to dark-colored moths decreased. Evolutionists
believe this to be a great piece of evidence for their theory. They
take refuge and solace in window-dressing, showing how light-colored
moths "evolved" into dark-colored ones.
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The top picture
shows trees with moths on them before the Industrial
Revolution, and the bottom picture shows them at
a later date. Because the trees had grown darker,
birds were able catch light-colored moths more easily
and their numbers decreased. However, this is not
an example of "evolution," because no new species
emerged; all that happened was that the ratio of
the two already existing types in an already existing
species changed.
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However, although we believe
these facts to be correct, it should be quite clear that they can
in no way be used as evidence for the theory of evolution, since
no new form arose that had not existed before. Dark colored moths
had existed in the moth population before the Industrial Revolution.
Only the relative proportions of the existing moth varieties in
the population changed. The moths had not acquired a new trait or
organ, which would cause "speciation."13 In order
for one moth species to turn into another living species, a bird
for example, new additions would have had to be made to its genes.
That is, an entirely separate genetic program would have had to
be loaded so as to include information about the physical traits
of the bird.
This is the answer to be given to the evolutionist
story of Industrial Melanism. However, there is a more interesting
side to the story: Not just its interpretation, but the story itself
is flawed. As molecular biologist Jonathan Wells explains in his
book Icons of Evolution, the story of the peppered moths, which
is included in every evolutionary biology book and has therefore,
become an "icon" in this sense, does not reflect the truth. Wells
discusses in his book how Bernard Kettlewell's experiment, which
is known as the "experimental proof" of the story, is actually a
scientific scandal. Some basic elements of this scandal are:
-Many experiments conducted after
Kettlewell's revealed that only one type of these moths rested on
tree trunks, and all other types preferred to rest beneath small,
horizontal branches. Since 1980 it has become clear that peppered
moths do not normally rest on tree trunks. In 25 years of fieldwork,
many scientists such as Cyril Clarke and Rory Howlett, Michael Majerus,
Tony Liebert, and Paul Brakefield concluded that in Kettlewell's
experiment, moths were forced to act atypically, therefore, the
test results could not be accepted as scientific.14
- Scientists who tested Kettlewell's conclusions came
up with an even more interesting result: Although the number of
light moths would be expected to be larger in the less polluted
regions of England, the dark moths there numbered four times as
many as the light ones. This meant that there was no correlation
between the moth population and the tree trunks as claimed by Kettlewell
and repeated by almost all evolutionist sources.
- As the research deepened, the scandal
changed dimension: "The moths on tree trunks" photographed by Kettlewell,
were actually dead moths. Kettlewell used dead specimens glued or
pinned to tree trunks and then photographed them. In truth, there
was little chance of taking such a picture as the moths rested not
on tree trunks but underneath the leaves.15
These facts were uncovered by the scientific community
only in the late 1990s. The collapse of the myth of Industrial Melanism,
which had been one of the most treasured subjects in "Introduction
to Evolution" courses in universities for decades, greatly disappointed
evolutionists. One of them, Jerry Coyne, remarked:
My own reaction resembles the dismay
attending my discovery, at the age of six, that it was my father
and not Santa who brought the presents on Christmas Eve.16
Thus, "the most famous example of natural selection"
was relegated to the trash-heap of history as a scientific scandal-which
was inevitable, because natural selection is not an "evolutionary
mechanism," contrary to what evolutionists claim.
In short, natural selection is capable neither of adding
a new organ to a living organism, nor of removing one, nor of changing
an organism of one species into that of another. The "greatest"
evidence put forward since Darwin has been able to go no further
than the "industrial melanism" of moths in England.
Why Natural Selection Can not Explain Complexity
As we showed at the beginning, the greatest problem
for the theory of evolution by natural selection, is that it cannot
enable new organs or traits to emerge in living things. Natural
selection cannot develop a species' genetic data; therefore, it
cannot be used to account for the emergence of new species. The
greatest defender of the theory of punctuated equilibrium, Stephen
Jay Gould, refer to this impasse of natural selection as follows;
The essence of Darwinism lies in
a single phrase: natural selection is the creative force of evolutionary
change. No one denies that selection will play a negative role in
eliminating the unfit. Darwinian theories require that it create
the fit as well.17
Another of the misleading methods that evolutionists
employ on the issue of natural selection is their effort to present
this mechanism as an intelligent designer. However, natural selection
has no intelligence. It does not possess a will that can decide
what is good and what is bad for living things. As a result, natural
selection cannot explain biological systems and organs that possess
the feature of "irreducible complexity." These systems and organs
are composed of a great number of parts cooperating together, and
are of no use if even one of these parts is missing or defective.
(For example, the human eye does not function unless it exists with
all its components intact).
Therefore, the will that brings all
these parts together should be able to foresee the future and aim
directly at the advantage that is to be acquired at the final stage.
Since natural selection has no consciousness or will, it can do
no such thing. This fact, which demolishes the foundations of the
theory of evolution, also worried Darwin, who wrote: "If it could
be demonstrated that any complex organ existed, which could not
possibly have been formed by numerous, successive, slight modifications,
my theory would absolutely break down."18
Mutations
A deformed foot, the product of mutation. |
Mutations are defined as breaks or replacements taking
place in the DNA molecule, which is found in the nuclei of the cells
of a living organism and which contains all its genetic information.
These breaks or replacements are the result of external effects
such as radiation or chemical action. Every mutation is an "accident,"
and either damages the nucleotides making up the DNA or changes
their locations. Most of the time, they cause so much damage and
modification that the cell cannot repair them.
Mutation, which evolutionists frequently hide behind,
is not a magic wand that transforms living organisms into a more
advanced and perfect form. The direct effect of mutations is harmful.
The changes effected by mutations can only be like those experienced
by people in Hiroshima, Nagasaki, and Chernobyl: that is, death,
disability, and freaks of nature…
The reason for this is very simple: DNA has a very
complex structure, and random effects can only damage it. Biologist
B. G. Ranganathan states:
First, genuine mutations are very
rare in nature. Secondly, most mutations are harmful since they
are random, rather than orderly changes in the structure of genes;any
random change in a highy ordered system will be for the worse, not
for the better. For example, if an earthquake were to shake a highly
ordered structure such as a building, there would be a random change
in the framework of the building, which, in all probability, would
not be an improvement.19
Not surprisingly, no useful mutation has been so far
observed. All mutations have proved to be harmful. The evolutionist
scientist Warren Weaver comments on the report prepared by the Committee
on Genetic Effects of Atomic Radiation, which had been formed to
investigate mutations that might have been caused by the nuclear
weapons used in the Second World War:
Many will be puzzled about the statement
that practically all known mutant genes are harmful. For mutations
are a necessary part of the process of evolution. How can a good
effect-evolution to higher forms of life-result from mutations practically
all of which are harmful?20
Every effort put into "generating a useful mutation"
has resulted in failure. For decades, evolutionists carried out
many experiments to produce mutations in fruit flies, as these insects
reproduce very rapidly and so mutations would show up quickly. Generation
upon generation of these flies were mutated, yet no useful mutation
was ever observed. The evolutionist geneticist Gordon Taylor writes
thus:
It is a striking, but not much mentioned
fact that, though geneticists have been breeding fruit-flies for
sixty years or more in labs all round the world- flies which produce
a new generation every eleven days-they have never yet seen the
emergence of a new species or even a new enzyme.21
Another researcher, Michael Pitman, comments on the
failure of the experiments carried out on fruit flies:
Morgan, Goldschmidt, Muller,
and other geneticists have subjected generations of fruit flies
to extreme conditions of heat, cold, light, dark, and treatment
by chemicals and radiation. All sorts of mutations, practically
all trivial or positively deleterious, have been produced. Man-made
evolution? Not really: Few of the geneticists' monsters could have
survived outside the bottles they were bred in. In practice mutants
die, are sterile, or tend to revert to the wild type.22
The same holds true for man. All mutations that have
been observed in human beings have had deleterious results. All
mutations that take place in humans result in physical deformities,
in infirmities such as mongolism, Down syndrome, albinism, dwarfism
or cancer. Needless to say, a process that leaves people disabled
or sick cannot be "an evolutionary mechanism"-evolution is supposed
to produce forms that are better fitted to survive.
The American pathologist David A.
Demick notes the following in a scientific article about mutations:
Since the beginning of the twentieth century,
evolutionary biologists have sought examples of useful mutations
by creating mutant flies. But these efforts have always
resulted in sick and deformed creatures. The top picture
shows the head of a normal fruit fly, and the picture on
the right shows the head of fruit fly with legs coming out
of it, the result of mutation. |
Literally thousands of human diseases associated with genetic mutations
have been catalogued in recent years, with more being described
continually. A recent reference book of medical genetics listed
some 4,500 different genetic diseases. Some of the inherited syndromes
characterized clinically in the days before molecular genetic analysis
(such as Marfan's syndrome) are now being shown to be heterogeneous;
that is, associated with many different mutations... With this array
of human diseases that are caused by mutations, what of positive
effects? With thousands of examples of harmful mutations readily
available, surely it should be possible to describe some positive
mutations if macroevolution is true. These would be needed not only
for evolution to greater complexity, but also to offset the downward
pull of the many harmful mutations. But, when it comes to identifying
positive mutations, evolutionary scientists are strangely silent.23
Mutant frogs born with crippled legs.
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The only instance evolutionary biologists give of "useful mutation"
is the disease known as sickle cell anemia. In this, the hemoglobin
molecule, which serves to carry oxygen in the blood, is damaged
as a result of mutation, and undergoes a structural change. As a
result of this, the hemoglobin molecule's ability to carry oxygen
is seriously impaired. People with sickle cell anemia suffer increasing
respiratory difficulties for this reason. However, this example
of mutation, which is discussed under blood disorders in medical
textbooks, is strangely
A mutant fly with deformed wings. |
evaluated by some evolutionary biologists as a "useful
mutation." They say that the partial immunity to malaria by those
with the illness is a "gift" of evolution. Using the same logic,
one could say that, since people born with genetic leg paralysis
are unable to walk and so are saved from being killed in traffic
accidents, therefore genetic leg paralysis is a "useful genetic
feature." This logic is clearly totally unfounded.
It is obvious that mutations are solely a destructive
mechanism. Pierre-Paul Grassé, former president of the French Academy
of Sciences, is quite clear on this point in a comment he made about
mutations. Grassé compared mutations to "making mistakes in the
letters when copying a written text." And as with mutations, letter
mistakes cannot give rise to any information, but merely damage
such information as already exists. Grassé explained this fact in
this way:
Mutations, in time, occur incoherently.
They are not complementary to one another, nor are they cumulative
in successive generations toward a given direction. They modify
what preexists, but they do so in disorder, no matter how…. As soon
as some disorder, even slight, appears in an organized being, sickness,
then death follow. There is no possible compromise between the phenomenon
of life and anarchy.24
So for that reason, as Grassé puts
it, "No matter how numerous they may be, mutations do not produce
any kind of evolution."25
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The shape and functions of red
corpuscles are compromised in sickle-cell anemia.
For this reason, their oxygen-carrying capacities
are weakened.
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The Pleiotropic Effect
The most important proof that mutations lead only to damage, is
the process of genetic coding. Almost all of the genes in a fully
developed living thing carry more than one piece of information.
For instance, one gene may control both the height and the eye color
of that organism. Microbiologist Michael Denton explains this characteristic
of genes in higher organisms such as human beings, in this way:
1. The wings do not develop.
2. The hind limbs reach full length, but the digits do
not fully develop.
3. There is no soft fur covering
4. Although there is a respiratory passage, lungs and
air sacs are absent.
5. The urinary tract does not grow, and does not induce
the development of the kidney.
On the left we can see the normal
development of a domesticated fowl, and on the right the
harmful effects of a mutation in the pleiotropic gene.
Careful examination shows that a mutation in just one
gene damages many different organs. Even if we hypothesize
that mutation could have a beneficial effect, this "pleiotropic
effect" would remove the advantage by damaging many more
organs.
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The effects of genes on development
are often surprisingly diverse. In the house mouse, nearly every
coat-colour gene has some effect on body size. Out of seventeen
x-ray induced eye colour mutations in the fruit fly Drosophila melanogaster,
fourteen affected the shape of the sex organs of the female, a characteristic
that one would have thought was quite unrelated to eye colour. Almost
every gene that has been studied in higher organisms has been found
to effect more than one organ system, a multiple effect which is
known as pleiotropy. As Mayr argues in Population, Species and Evolution:
"It is doubtful whether any genes that are not pleiotropic exist
in higher organisms."26
Because of this characteristic of the genetic structure
of living things, any coincidental change because of a mutation,
in any gene in the DNA, will affect more than one organ. Consequently,
this mutation will not be restricted to one part of the body, but
will reveal more of its destructive impact. Even if one of these
impacts turns out to be beneficial, as a result of a very rare coincidence,
the unavoidable effects of the other damage it causes will more
than outweigh those benefits.
To summarize, there are three main reasons why mutations
cannot make evolution possible:
l- The direct effect of mutations is harmful: Since
they occur randomly, they almost always damage the living organism
that undergoes them. Reason tells us that unconscious intervention
in a perfect and complex structure will not improve that structure,
but will rather impair it. Indeed, no "useful mutation" has ever
been observed.
2- Mutations add no new information to an organism's
DNA: The particles making up the genetic information are either
torn from their places, destroyed, or carried off to different places.
Mutations cannot make a living thing acquire a new organ or a new
trait. They only cause abnormalities like a leg sticking out of
the back, or an ear from the abdomen.
3- In order for a mutation to be transferred to the
subsequent generation, it has to have taken place in the reproductive
cells of the organism: A random change that occurs in a cell or
organ of the body cannot be transferred to the next generation.
For example, a human eye altered by the effects of radiation, or
by other causes, will not be passed on to subsequent generations.
The Escherichia coli bacterium
is no different from specimens a billion years old. Countless
mutations over this long period have not led to any structural
changes. |
All the explanations provided above indicate that natural
selection and mutation have no evolutionary effect at all. So far,
no observable example of "evolution" has been obtained by this method.
Sometimes, evolutionary biologists claim that "they cannot observe
the evolutionary effect of natural selection and mutation mechanisms
since these mechanisms take place only over an extended period of
time." However, this argument, which is just a way of making themselves
feel better, is baseless, in the sense that it lacks any scientific
foundation. During his lifetime, a scientist can observe thousands
of generations of living things with short life spans such as fruit
flies or bacteria, and still observe no "evolution." Pierre-Paul
Grassé states the following about the unchanging nature of bacteria,
a fact which invalidates evolution:
Bacteria ...are the organisms
which, because of their huge numbers, produce the most mutants.
[B]acteria ...exhibit a great fidelity to their species. The bacillus
Escherichia coli, whose mutants have been studied very carefully,
is the best example. The reader will agree that it is surprising,
to say the least, to want to prove evolution and to discover its
mechanisms and then to choose as a material for this study a being
which practically stabilized a billion years ago! What is the use
of their unceasing mutations, if they do not [produce evolutionary]
change? In sum, the mutations of bacteria and viruses are merely
hereditary fluctuations around a median position; a swing to the
right, a swing to the left, but no final evolutionary effect. Cockroaches,
which are one of the most venerable living insect groups, have remained
more or less unchanged since the Permian, yet they have undergone
as many mutations as Drosophila, a Tertiary insect.27
Briefly, it is impossible for living beings to have
evolved, because there exists no mechanism in nature that can cause
evolution. Furthermore, this conclusion agrees with the evidence
of the fossil record, which does not demonstrate the existence of
a process of evolution, but rather just the contrary.
7 Charles
Darwin, The Origin of Species by Means of Natural Selection, The
Modern Library, New York, p. 127. (emphasis added)
8 V. C. Wynne-Edwards, "Self Regulating
Systems in Populations of Animals, Science, vol. 147, 26 March 1965,
pp. 1543-1548; V. C. Wynne-Edwards, Evolution Through Group Selection,
London, 1986.
9 A. D. Bradshaw, "Evolutionary significance
of phenotypic plasticity in plants," Advances in Genetics, vol.
13, pp. 115-155; cited in Lee Spetner, Not By Chance!: Shattering
the Modern Theory of Evolution, The Judaica Press, Inc., New York,
1997, pp. 16-17.
10 Andy Coghlan "Suicide Squad", New
Scientist, 10 July 1999.
11 Colin Patterson, "Cladistics", Interview
by Brian Leek, interviewer Peter Franz, March 4, 1982, BBC.(emphasis
added)
12 Phillip E. Johnson, Darwin On Trial,
Intervarsity Press, Illinois, 1993, p. 27.
13 For more detailed information about
Industrial Melanism, please see Phillip Johnson, Darwin on Trial,
InterVarsity Press, 2nd. Ed., Washington D.C., p. 26.
14 Jonathan Wells, Icons of Evolution: Science
or Myth? Why Much of What We Teach About Evolution is Wrong, Regnery
Publishing, Washington, 2000, pp. 149-150.
15 Jonathan Wells, Icons of Evolution:
Science or Myth? Why Much of What We Teach About Evolution is Wrong,
Regnery Publishing, Washington, 2000, pp. 141-151.
16 Jerry Coyne, "Not Black and White",
a review of Michael Majerus's Melanism: Evolution in Action, Nature,
396, 1988, pp. 35-36.
17 Stephen Jay Gould, "The Return of
Hopeful Monster", Natural History, vol. 86, June-July 1977, p. 28.
18 Charles Darwin, The Origin of Species:
A Facsimile of the First Edition, Harvard University Press, 1964,
p. 189.(emphasis added)
19 B. G. Ranganathan, Origins?, Pennsylvania:
The Banner Of Truth Trust, 1988. (emphasis added)
20 Warren Weaver et al., "Genetic Effects
of Atomic Radiation", Science, vol. 123, June 29, 1956, p. 1159.
(emphasis added)
21 Gordon Rattray Taylor, The Great
Evolution Mystery, Abacus, Sphere Books, London, 1984, p. 48.
22 Michael Pitman, Adam and Evolution, River
Publishing, London, 1984, p. 70. (emphasis added)
23 David A. Demick,
"The Blind Gunman", Impact, no. 308, February 1999. (emphasis added)
24 Pierre-Paul Grassé, Evolution
of Living Organisms, Academic Press, New York, 1977, p. 97, 98.
25 Pierre-Paul Grassé, Evolution of
Living Organisms, Academic Press, New York, 1977, p. 88. (emphasis
added)
26 Michael Denton, Evolution: A Theory in Crisis,
Burnett Books Ltd., London, 1985, p. 149.
27 Pierre-Paul Grassé, Evolution of Living Organisms,
Academic Press, New York, 1977, p. 87. (emphasis added) |